There are many different types of evidence. Certainly one that wasn't explored in the CSAS was the sea lice. Simon Jones talked about that briefly here. Certainly there are challenges with resistance to the drug and the ability of the industry to keep the lice levels down.
In terms of the work that we've done, our evidence centres around two pathogens—PRV and Tenacibaculum—but we're not done. Those are the ones that we've actually performed the models on to look at those risks. There very well may be others.
One thing we are embarking on now is looking at environmental DNA, which is a way of looking at the concentration of infective agents in the water column. We will actually be able to look at whether or not we can recognize the shift in the pathogen communities in the water column when farms are switching to a disease state before that disease state causes significant mortality. This would be potentially a tool that the regulator could use to determine what kind of mitigative actions they could take before there's major loss or before there is more risk to wild salmon.